What is the dosing strategy for alteplase for pulmonary embolism-induced cardiac arrest and what is the evidence to support that strategy?

Comment by InpharmD Researcher

In pulmonary embolism-induced cardiac arrest, alteplase dosing strategies vary widely because the evidence base is low quality and relies largely on observational studies, registries, and case series. Although the FDA-labeled regimen for acute massive pulmonary embolism (PE) is 100 mg IV (15 mg bolus followed by 85 mg over 2 hours), in cardiac arrest many protocols most commonly use a 50 mg IV bolus during ongoing CPR, sometimes with repeat bolus dosing if ROSC is not achieved, and alternative regimens include accelerated or infusion-based strategies. Guideline statements support considering thrombolysis when PE is suspected or confirmed and emphasize continuing CPR for 60-90 minutes after fibrinolysis. Overall, outcomes data are mixed and PE-arrest-specific comparative trials are limited, so dosing remains practice-variable and should be individualized while balancing potential benefit against bleeding risk.

pulmonary embolism-induced cardiac arrest, cardiac arrest; alteplase, tissue plasminogen activator, tPA, thrombolytic

Background

The 2025 American Heart Association (AHA) Guidelines reviewed the use of systemic thrombolysis for cardiac arrest caused by suspected or confirmed pulmonary embolism, drawing primarily from observational studies, registry data, case series, and limited randomized trials. Alteplase is most commonly administered as a 50 mg intravenous bolus during ongoing cardiopulmonary resuscitation, with some protocols permitting a second 50 mg bolus if return of spontaneous circulation (ROSC) is not achieved; alternative regimens include 100 mg infused over 2 hours. The guidelines emphasize the importance of prolonged CPR following thrombolytic administration, with supporting data and European Society of Cardiology guidance recommending resuscitation efforts continue for at least 60 to 90 minutes after fibrinolysis. Registry-based studies suggest improved short-term survival in PE-related cardiac arrest when thrombolysis is used, although randomized trials in unselected cardiac arrest populations have not demonstrated a mortality benefit. Based on these findings, alteplase may be a reasonable option in PE-induced cardiac arrest, particularly when used with rapid bolus dosing and extended resuscitation, although the overall quality of evidence remains low and further PE-specific studies are needed. [1]

The 2021 European Resuscitation Council Advanced Life Support guidelines do not specify an alteplase dose for pulmonary embolism–related cardiac arrest but recommend considering thrombolytic therapy when pulmonary embolism is suspected or confirmed and continuing cardiopulmonary resuscitation for 60-90 minutes after administration. This guidance is based on very low-certainty evidence summarized by the 2020 International Liaison Committee on Resuscitation, which showed no neurologic benefit, mixed survival outcomes, and inconsistent effects on return of spontaneous circulation. Consequently, thrombolytic use carries a weak recommendation, as potential benefit may outweigh bleeding risk despite limited and inconsistent data. [2]

The 2003 British Thoracic Society (BTS) guidelines recommend systemic thrombolysis as first-line therapy for massive pulmonary embolism, including cases where cardiac arrest is imminent or occurring, and state that a 50 mg intravenous bolus of alteplase may be administered during cardiopulmonary resuscitation. This dosing approach is supported by limited evidence, primarily consisting of case reports and small studies, as large randomized trials in patients with massive or arrest-related pulmonary embolism are not feasible. The guidelines cite reports of return of spontaneous circulation within minutes of alteplase administration during CPR and acknowledge that survival, while uncommon, has been documented in otherwise fatal presentations. Given the extremely high mortality associated with massive pulmonary embolism and circulatory collapse, the BTS concludes that empiric alteplase bolus dosing is reasonable despite low quality evidence, as the potential life-saving benefit is judged to outweigh the risk of bleeding in this setting. [3]

A 2021 publication evaluated the use of recombinant tissue plasminogen activator (rtPA), specifically alteplase, in the context of circulatory arrest induced by pulmonary embolism (PE). This study contended that while current guidelines recommend a 100 mg/2 h regimen of rtPA for hemodynamic instability, this approach is not suitable for circulatory arrest scenarios owing to altered pharmacokinetics during cardiopulmonary resuscitation (CPR). The authors argued in favor of an accelerated regimen cited in the 2019 ESC/ERS Guideline of 0.6 mg/kg over 15 minutes, with a maximum dose of 50 mg, based on the rationale that the low-flow state during CPR affects the metabolism and distribution of alteplase, extending its half-life. To support this perspective, the authors conducted a thorough review of the existing literature, identifying limited studies directly comparing the accelerated regimen to standard care in circulatory arrest scenarios. Retrospective analyses, such as the 2016 PEAPETT trial, demonstrated high rates of return of spontaneous circulation (ROSC) and survival using the accelerated regimen without significant bleeding complications. In patients with massive or submassive PE without circulatory arrest, the accelerated regimen showed a trend toward fewer bleeding events compared to the two-hour regimen, as noted in a meta-analysis including a 2010 study by Wang et al. These findings collectively provide a compelling argument for using the accelerated alteplase infusion in the unique setting of PE-induced circulatory arrest. [4]

The use of thrombolysis during cardiac arrest is typically performed when there is a known or highly suspected etiology of thrombosis causing the arrest. Yet there is great variability in the use and dose of thrombolytic agents. In a 2010 article, two controlled clinical studies were identified that used alteplase for resuscitation in cardiac arrest. However, neither cases strictly observed a 100 mg intravenous (IV) push dose. The first study used alteplase administered as 50 mg IV push followed by 5000 U of heparin in cardiac arrest patients who failed standard resuscitation, while the second study administered alteplase 100 mg over 15 minutes for out-of-hospital cardiac arrest patients. IV push is typically defined as administration over 5 minutes so while the 15 minute dose rate is shorter than usual, it may not be considered an IV push. Furthermore, neither studies were able to report a statistically significant mortality benefit. Although the first study reported more patients attaining return to spontaneous circulation in the alteplase plus heparin group (68% vs 44%, p= 0.026), patient survival 24 hours after the arrest was not significantly different compared to control (35% vs 22%; p= 0.171). Similarly, the second study only observed 1 of the 233 patients enrolled in the study surviving to discharge. Nevertheless, the authors of the review still suggest thrombolysis may be beneficial if pulmonary embolism-induced cardiac arrest is suspected, followed by 15 minutes of continued cardiac pulmonary resuscitation to allow time for the medication to act. [5], [6], [7]

In a 2019 retrospective cohort study, a small number of patients were enrolled to characterize the administration method of alteplase for presumed or confirmed pulmonary embolism during cardiac arrest. Patients received alteplase either by bolus only (mean bolus dose 53.3 mg; mean cumulative dose 62.5), infusion only (mean infusion dose 87.5 mg; mean cumulative dose 76.3 mg), or bolus with infusion administration (mean bolus dose 32.7 mg; mean infusion dose 64.5 mg; mean cumulative dose 99 mg). In the bolus-only group, the majority of patients received alteplase 50 mg (range 15-100 mg); limited details are available regarding the patients who received alteplase 100 mg bolus. The mean time from cardiac arrest to onset of alteplase administration was 15.1 minutes in the bolus-only group, 46.4 minutes in the infusion-only group, and 48 minutes in the bolus with infusion group (p= 0.006). Only 2 of 16 patients in the bolus group survived to hospital discharge compared to 1 of 8 patients in the infusion-only group and 8 of 11 patients in the bolus with infusion group. There was no indication that a 100 mg bolus dose was more effective than other regimens. However, cumulative doses of alteplase were found to be significantly higher in patients who returned to spontaneous circulation (ROSC), but the small patient population limits the reliability of the results. Within their discussion, the authors note the lack of specific guideline recommendations for thrombolytic dosing, while the literature reports various dosing strategies without a clear consensus for the best dosing strategy. [8]

A multicenter, retrospective study published in 2021 evaluated the use of thrombolysis in cardiac arrest secondary to suspected or confirmed pulmonary embolism (PE). Of 47 patients who were identified to have an order for alteplase or tenecteplase, 20 were excluded due to post-ROSC thrombolysis. Thrombolytic therapy was utilized in 27 patients for pericardiac arrest with a suspected or confirmed PE. Of 11 patients who achieved ROSC, the most common dosing strategy was alteplase 100 mg (5 of 11 patients) and alteplase 50 mg (5 of 11 patients); the remaining patient received tenecteplase. It was specified that no patients received sequential boluses following the initial bolus. The authors discuss a previous international survey of thrombolytic use in cardiac arrest secondary to PE in which the most common regimen was alteplase 50 mg IV push, with the ability to repeat in 10 to 30 minutes if ROSC was not achieved. However, the survey does not describe whether an additional bolus was given following the achievement of RSOC. [9], [10]

References: [1] Cao D, Arens AM, Chow SL, et al. Part 10: Adult and Pediatric Special Circumstances of Resuscitation: 2025 American Heart Association Guidelines for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care. Circulation. 2025;152(suppl 2):S578-S672. doi:10.1161/CIR.0000000000001380
[2] Soar J, Böttiger BW, Carli P, et al. European Resuscitation Council Guidelines 2021: Adult Advanced Life Support. Resuscitation. 2021;161:115-151. doi:10.1016/j.resuscitation.2021.02.010
[3] British Thoracic Society Standards of Care Committee. Guidelines for the management of suspected acute pulmonary embolism. Thorax. 2003;58(6):470-484. doi:10.1136/thorax.58.6.470
[4] Bakkum MJ, Schouten VL, Smulders YM, Nossent EJ, van Agtmael MA, Tuinman PR. Accelerated treatment with rtPA for pulmonary embolism induced circulatory arrest. Thromb Res. 2021;203:74-80. doi:10.1016/j.thromres.2021.04.023
[5] Perrott J, Henneberry RJ, Zed PJ. Thrombolytics for cardiac arrest: case report and systematic review of controlled trials. Ann Pharmacother. 2010;44(12):2007-2013. doi:10.1345/aph.1P364
[6] Böttiger BW, Bode C, Kern S, et al. Efficacy and safety of thrombolytic therapy after initially unsuccessful cardiopulmonary resuscitation: a prospective clinical trial. Lancet. 2001;357(9268):1583-1585. doi:10.1016/S0140-6736(00)04726-7
[7] Abu-Laban RB, Christenson JM, Innes GD, et al. Tissue plasminogen activator in cardiac arrest with pulseless electrical activity [published correction appears in N Engl J Med. 2003 Oct 9;349(15):1487]. N Engl J Med. 2002;346(20):1522-1528. doi:10.1056/NEJMoa012885
[8] Peppard SR, Parks AM, Zimmerman J. Characterization of alteplase therapy for presumed or confirmed pulmonary embolism during cardiac arrest. Am J Health Syst Pharm. 2018;75(12):870-875. doi:10.2146/ajhp170450
[9] Kataria V, Kohman K, Jensen R, Mora A. Usefulness of thrombolysis in cardiac arrest secondary to suspected or confirmed pulmonary embolism. Proc (Bayl Univ Med Cent). 2021;34(4):442-445. Published 2021 Apr 20. doi:10.1080/08998280.2021.1911494
[10] Rech MA, Horng M, Holzhausen JM, et al. International Survey of Thrombolytic Use for Treatment of Cardiac Arrest Due to Massive Pulmonary Embolism. Crit Care Explor. 2020;2(6):e0132. Published 2020 Jun 9. doi:10.1097/CCE.0000000000000132
Literature Review

A search of the published medical literature revealed 10 studies investigating the researchable question:

What is the dosing strategy for alteplase for pulmonary embolism-induced cardiac arrest and what is the evidence to support that strategy?

Level of evidence

B - One high-quality study or multiple studies with limitations  Read more→



Please see Tables 1-10 for your response.


Half-Dose versus Full-Dose Alteplase for Treatment of Pulmonary Embolism

Design

Retrospective cohort study

N= 3768

Objective

To evaluate the effectiveness and safety of half-dose versus full-dose alteplase for the treatment of pulmonary embolism

Study Groups

Half-Dose (n= 699)

Full-Dose (n= 3069)

Inclusion Criteria

Age ≥18 years, admitted to the ICU on hospital day 1 or 2 with primary diagnosis of pulmonary embolism, treated with alteplase 50 or 100 mg IV.

Exclusion Criteria

Received alteplease doses aside from the listed amount, receiving catheter thrombus fragmentation as first intervention for pulmonary embolism.

Methods

Patient data was assessed based on those who received alteplase 50 mg versus 100 mg IV for the treatment of pulmonary embolism. Alteplease 50 mg is considered half-dose while alteplase 100 mg is considered full-dose. An analysis of included patients was performed as well as those who underwent propensity score matching.

Duration

January 2010 - December 2014

Outcome Measures

Primary Outcome: Treatment Escalation 

Secondary Outcome: Hospital Mortality, Readmission for Pulmonary Embolism (PE), ICU Length of stay

Safety Outcomes: Major bleeding, Documentation of fibrinolytic therapy-related Adverse Event

Second analysis was performed for propensity matched cohorts.

Baseline Characteristics

 

Alteplase 50 mg (n= 699)

Alteplase 100 mg (n= 3069)

 

Age >65

247 (35.3%)  1126 (36.7%)   

Female

336 (48.1%)  1543 (50.3%)   

White

486 (69.5%)  2123 (69.2%)   

Hypertension

378 (54.1%) 1859 (60.6%)  

Venous Thromboembolism

443 (63.4%) 1552 (50.6%)  

Results

Endpoint

Alteplase 50 mg (n= 699)

Alteplase 100 mg (n= 3069)

p-value

Escalation of Treatment

379 (54.2%)  1442 (47.2%)  <0.01 

Readmitted for PE

41 (5.9%) 252 (8.2%)  0.04

In-hospital Mortality

82 (11.7%) 656 (21.4%) <0.01

ICU Length of Stay, days

2 (1-4) 2 (1-4) 0.18

Cerebral Hemorrhage

0.6% 0.5% 0.70

GI Bleed

1.9% 2.2% 0.58

Fibrinolytic Causing Adverse Event

1.7% 2.0% 0.34
  Matched alteplase 50 mg (n=548) Matched alteplase 100 mg (n=548) p-value

Escalation of Treatment

295 (53.8%) 227 (41.4%) <0.01

Readmitted for PE

35 (6.4%) 40 (7.3%) 0.55

In Hospital Mortality

71 (13.0%) 83 (15.1%) 0.30

Adverse Events

As shown in the results section, bleeding is always a risk when on fibrinolytic or anticoagulatory therapy, but there were no differences seen between the two treatment arms as far as adverse effects are concerned.

Study Author Conclusions

Compared to full-dose alteplase, half-dose was associated with similar mortality and rates of major bleeding. Treatment escalation occurred more often in half-dose treated patients. These results question whether half-dose alteplase provides similar efficacy with improved safety, and highlights the need for further study before use of half-dose alteplase therapy can be routinely recommended in patients with PE.

InpharmD Researcher Critique

There are a few limitations in this study given the retrospective design of the study. The authors were unable to always determine why a particular thrombolytic strategy was given based on treatment protocols. In addition patient body weights were unavailable so the assessment of weight-based dosing was impossible. Despite this, the study had a large sample size and rigorous statistical analyses were done to minimize bias.

References:
[1] Kiser TH, Burnham EL, Clark B, et al. Half-Dose Versus Full-Dose Alteplase for Treatment of Pulmonary Embolism. Crit Care Med. 2018;46(10):1617-1625. doi:10.1097/CCM.0000000000003288

Pulseless Electrical Activity in Pulmonary Embolism Treated with Thrombolysis, (From the “PEAPETT” Study)

Design

Retrospective cohort study

N= 23

Objective

To assess the effects of low dose tissue plasminogen activator (tPA) on the clinical and echocardiographic outcomes of patients who had presented with a pulseless electrical activity (PEA) and cardiopulmonary arrest due to confirmed pulmonary embolism (PE)

Study Groups

Study participants (N= 23)

Inclusion Criteria

Developed cardiopulmonary arrest with PEA, received cardiopulmonary resuscitation (CPR) due to massive PE

Exclusion Criteria

N/A

Methods

Surviving patients who filled the criteria were retrospectively identified to be prospectively followed in this cohort study. Patients would be seen in office within three weeks of discharge and then 6 months thereafter. Venous duplex scans were performed for suspicion of deep vein thrombosis (DVT). Pulmonary CT angiography or V/Q scanning was performed if PE was suspected.

Patients received 50 mg alteplase (Activase®) IV push over one minute during ongoing CPR followed by 10 mL of saline. The 100 mg alteplase bottles were mixed with 100 mL of sterile water, then half was drawn into a 50 mL syringe. Heparin 2000 to 5000 IU was subsequently given as a bolus, along with a maintenance drip starting at 10 U/kg/hour. The activated partial thromboplastin time (aPTT) goal for heparin was between 60 to 100 seconds. After 24 to 30 hours, heparin was discontinued and patients were started on apixaban or rivaroxaban PO at maintenance dose.

Duration

Enrollment period: February 2013 to December 2015

Follow-up: 22 ± 3 months

Outcome Measures

Return of spontaneous circulation

Baseline Characteristics

 

Study participants (N= 23)

Age, years

72

Men

9 (39%)

Body mass index, kg/m2

35

Previous or concomitant disease

Hypertension

Diabetes mellitus

Cardiovascualr disease

Hypercholesterolemia

Pulmonary disease

Renal disease

 

15 (65%)

10 (43%)

7 (30%)

13 (57%)

14 (61%)

5 (22%)

Current smoker

10 (43%)

Unprovoked PE

16 (70%)

Known prothrombotic state

6 (26%)

Previous venous thromboembolism (VTE)

7 (30%)

Cancer

Active

History

 

3 (13%)

5 (22%)

Concomitant DVT

15 (65%)

Results

Endpoint

Study participants (N= 23)

Return of spontaneous circulation within 2 to 15 minutes

22

Minor/major bleeding

0

Recurrent VTE or bleeding during hospitalization/follow-up

0

Deaths

3*

*Two patients died in the hospital. One due to irreversible shock after 10 hours of admission and another due to permanent hypoxemic encephalopathy which led to multisystem organ failure 8 days later. One patient died due to cancer during the follow-up period.

Study Author Conclusions

Rapid administration of 50 mg of tPA is safe and effective in restoration of spontaneous circulation in PEA due to massive PE leading to enhanced survival and a significant reduction in pulmonary artery pressures.

InpharmD Researcher Critique

This cohort study was primarily a retrospective, observational study without a control group to assess the magnitude of alteplase's benefit. The outcomes of the study are speculative at best without comparison data.
References:
[1] Sharifi M, Berger J, Beeston P, et al. Pulseless electrical activity in pulmonary embolism treated with thrombolysis (from the "PEAPETT" study). Am J Emerg Med. 2016;34(10):1963-1967. doi:10.1016/j.ajem.2016.06.094

Characterization of alteplase therapy for presumed or confirmed pulmonary embolism during cardiac arrest

Design

Multicenter, retrospective, cohort study

N= 35

Objective

To characterize the dosing and administration of alteplase in cardiac arrest due to suspected or confirmed pulmonary embolism (PE) 

Study Groups

Bolus only (n= 16)

Infusion only (n= 8)

Bolus with infusion (n= 11)

Inclusion Criteria

Aged ≥ 18 years, received alteplase intravenous (IV) for confirmed or presumed massive PE during cardiac arrest

Exclusion Criteria

N/A

Methods

Patient data that fulfilled the criteria and involved the dispensing of alteplase exceeding 2 mg were analyzed.

Confirmed PE required a definitive image via screening (e.g., computed tomography). Presumed PE involved either bedside echocardiography findings or the physician's clinical judgement.

Duration

Data collection period: November 1, 2010 to February 1, 2015

Outcome Measures

Primary: characterizing the use of alteplase in the setting of cardiac arrest

Secondary: return to spontaneous circulation, surviving cardiac arrest, surviving hospital discharge, ICU length of stay

Baseline Characteristics

 

Bolus only (n= 16)

Infusion only (n= 8)

Bolus with infusion (n= 11)

Age, years

59 57 55

Men

10 4 5

Initial cardiac rhythm during arrest

Pulseless electrical activity

Other, shockable rhythm

Other, non-shockable rhythm

 

11

0

5

 

3

1

4

 

9

0

2

Admission diagnosis

Cardiac arrest

Pulmonary embolism (PE)

Deep vein thrombosis (DVT)

Other

 

6

2

0

5

 

6

0

0

2

 

2

3

1

1

Medical history

Malignancy

Immobilization or recent surgery

Previous DVT or PE

 

4

1

2

 

1

2

3

 

6

2

2

Results

Endpoint

Bolus only (n= 16)

Infusion only (n= 8)

Bolus with infusion (n= 11)

Characteristic of alteplase

Bolus dose, mg

Infusion dose, mg

Cumulative dose, mg

Total administration time, min

Time after arrest to administration, min

 

53.3

--

62.5

--

15.1

 

--

87.5

76.3

97.5

46.4

 

32.7

64.5

99

96.3

48.8

Clinical outcomes

Time to return of spontaneous circulation (ROSC), min

Surviving cardiac arrest

Surviving to hospital discharge

ICU length of stay, days

Hospital length of stay, days

 

6

24.0

2

2.8

3.9

 

3

12.3

1

0

1.0

 

8

32.3

8

3

2.9

Adverse Events

Two major bleeding events occurred, both in patients who received bolus-with-infusion alteplase and had ROSC. Three minor bleeding events occurred among patients in the other 2 groups; of these patients, two did not have ROSC, and one did.

Alteplase bolus doses in patients who had bleeding ranged from 20 to 100 mg. Both patients with major bleeding received a cumulative alteplase dose of 100 mg; both survived the cardiac arrest, but only one survived to hospital discharge.

Study Author Conclusions

Among patients receiving alteplase for presumed or confirmed PE during cardiac arrest, the most common treatment was administration of a single 50-mg bolus of the thrombolytic agent. This treatment was received by all survivors of cardiac arrest.

InpharmD Researcher Critique

This was a study focused on characterizing the use of alteplase which does not illustrate the benefit of treatment compared to similar patients who did not receive alteplase. 
References:
[1] Peppard SR, Parks AM, Zimmerman J. Characterization of alteplase therapy for presumed or confirmed pulmonary embolism during cardiac arrest. Am J Health Syst Pharm. 2018;75(12):870-875. doi:10.2146/ajhp170450

Central Venous Catheter-directed Tissue Plasminogen Activator in Massive Pulmonary Embolism

Design

Case report

Case presentation

An 88-year-old obese female with sudden cardiac arrest with hypoxia, persistent hypotension, and suspected massive pulmonary embolism (PE) causing respiratory failure presented to the emergency department (ED). She was intubated and sedated with a heart rate of 92 beats per minute (bpm), blood pressure 53/40 mmHg, respiratory rate 21 bpm by manual bag, and oxygen saturation of 88% on 100% (FiO2). Electrocardiogram (ECG) revealed a right bundle branch block without signs of acute myocardial infarction. In addition to a peripheral norepinephrine infusion, a 1 mg/min of 50 milligrams tPA infusion was initiated without any significant response to the thrombolysis. Cardiopulmonary resuscitation (CPR) was initiated. A 50 mg push tPA bolus through a central venous catheter (CVC) was also administered. Return of spontaneous circulation (ROSC) was achieved after CPR continued for four additional minutes. Following a total of 1 mg of epinephrine and 75 milliequivalents of bicarbonate, patient’s hemodynamics improved and she was admitted to the medical intensive care unit and eventually discharged home on hospital day 13 on oral anticoagulation (apixaban).

The tPA infusion was implied but not defined as alteplase.

Study Author Conclusions

The resulting hemodynamic collapse in this elderly female was thought to be successfully treated with CVC-directed tPA. The patient had a return of cardiac function, resolution of hypoxia, and ultimately had a favorable outcome. Patients in cardiac arrest or peri-arrest in the setting of PE may benefit significantly from a bolus of tissue plasminogen activator through a central venous catheter. In the future, more evidence is needed to analyze CVC-directed tPA and its effect on morbidity and mortality.

References:
[1] Gulati V, Brazg J. Central Venous Catheter-directed Tissue Plasminogen Activator in Massive Pulmonary Embolism. Clin Pract Cases Emerg Med. 2018;2(1):67-70. Published 2018 Jan 18. doi:10.5811/cpcem.2017.11.35845

Evaluation of Rescue Thrombolysis in Cardiac Arrest Secondary to Suspected or Confirmed Pulmonary Embolism

Design

Single-center, retrospective chart review

N= 22

Objective

To describe clinical outcomes among patients who received rescue thrombolytic therapy during a cardiac arrest for suspected or confirmed pulmonary embolism (PE)

Study Groups

Study patients (N= 22)

Inclusion Criteria

Age 18 years or older, received alteplase or tenecteplase, cardiac arrest secondary to suspected or confirmed PE

Exclusion Criteria

N/A

Methods

Data were collected from a single academic medical center and analyzed to determine relevant endpoints including survival, the incidence of return to spontaneous circulation (ROSC) after thrombolytic administration, risk of bleeding, and length of hospital stay. ROSC was defined as the achievement of spontaneous circulation at any point after thrombolytic administration. Bleeding was assessed up to 7 days after thrombolytic administration or until patient death.

Duration

7 days or until the patient death

Outcome Measures

Primary: survival to hospital discharge in patients who received alteplase 100 mg, intensive care unit (ICU) length of stay, hospital length of stay

Secondary: achieved ROSC in patients who received alteplase 100 mg, experienced bleeding

Baseline Characteristics

 

Study patients (N= 22)

Age, years

66

Female

10

Weight, kg

80.3

In-hospital cardiac arrest

15

Pulseless electrical activity at initial arrest

12

Confirmed pulmonary embolism prior to arrest

3

Thrombolytic push strategy

Alteplase 100 mg

Alteplase 75 mg

Alteplase 50 mg

Alteplase 50 mg + 50 mg

Alteplase 20 mg

Tenecteplase 50 mg

Tenecteplase 45 mg

Tenecteplase 40 mg

 

13

1

1

2

1

1

2

1

Results

Endpoint

Study patients (N= 22)

Survival to hospital discharge on alteplase 100 mg

ICU length of stay, days

Hospital length of stay, days

1 of 3

7.9

21.1

Achieved ROSC on alteplase 100 mg

6/13

Bleeding events on alteplase 100 mg

0

Study Author Conclusions

Medical advances in PE management continue to evolve; yet the role of thrombolytic therapy in PE-related cardiac arrest remains unclear, with low overall rates of survival. These findings add to the relatively small body of evidence and highlight that optimal dosing remains unknown in this setting.

InpharmD Researcher Critique

The results of the study are exploratory in nature. There are limited details on the IV push regimen of thrombolytic (unknown duration of administration).
References:
[1] Summers K, Schultheis J, Raiff D, Dahhan T. Evaluation of Rescue Thrombolysis in Cardiac Arrest Secondary to Suspected or Confirmed Pulmonary Embolism. Ann Pharmacother. 2019;53(7):711-715. doi:10.1177/1060028019828423

 

Retrospective review of thrombolytic use for cardiac arrest due to suspected pulmonary embolism

Design

Retrospective, descriptive cohort study

N= 32

Objective

To describe the characteristics and outcomes of patients who received a thrombolytic agent during cardiac arrest due to suspected PE, including efficacy and safety

Study Groups

Alteplase group (n= unknown)

Tenecteplase group (n= unknown)

Inclusion Criteria

Adult patients who received alteplase or tenecteplase in the emergency department during active cardiac arrest

Exclusion Criteria

Not specified

Methods

Retrospective review of 32 adult patients who received alteplase or tenecteplase during cardiac arrest in the emergency department. Agent selection and dosing were at the discretion of the primary provider. The median dose for alteplase was 50 mg and for tenecteplase was 45 mg.

Duration

Not specified

Outcome Measures

Primary: Achievement of return of spontaneous circulation (ROSC)

Secondary: Survival to hospital admission, survival to hospital discharge, incidence of major bleeding events

Baseline Characteristics  

All patients (n= 32)

Mean age, years 63
Results  

Alteplase group

Tenecteplase group
ROSC achieved

9

2
Survived to hospital admission

5

2
Survived to hospital discharge

1

1
Adverse Events

All but one patient experienced a major bleeding event during admission.

Study Author Conclusions

This study provides new data regarding the outcomes of thrombolytic therapy in patients experiencing cardiac arrest due to suspected massive PE. Despite administration of thrombolytics, survival to hospital admission and subsequent survival to hospital discharge were seen in only a very small proportion of patients. Further research is necessary to optimize the management of this life-threatening condition.

Critique

The study provides valuable insights into the use of thrombolytics in cardiac arrest due to suspected PE, but is limited by its small sample size and retrospective design. The lack of a control group and the discretionary dosing by providers may introduce bias and limit the generalizability of the findings.

 

References:
[1] Jansen A, Schmitt CJ, Cabrera D, Wieruszewski ED. Retrospective review of thrombolytic use for cardiac arrest due to suspected pulmonary embolism. Am J Emerg Med. 2025;94:76-80. doi:10.1016/j.ajem.2025.04.036

 

Double Bolus Alteplase Therapy during Cardiopulmonary Resuscitation for Cardiac Arrest due to Massive Pulmonary Embolism Guided by Focused Bedside Echocardiography

Design

Case report

Case presentation

A 56-year-old female who had undergone knee replacement surgery two weeks prior, presented with acute respiratory distress and subsequently experienced cardiac arrest in the emergency department. During CPR, a focused bedside echocardiogram was utilized, revealing a severely dilated right ventricle indicative of a massive pulmonary embolism.

An initial bolus of 50 mg of tissue plasminogen activator (t-PA) was administered intravenously, leading to return of spontaneous circulation (ROSC). However, persistent hemodynamic instability prompted the administration of a second 50 mg bolus of t-PA 20 minutes later, guided by ongoing echocardiographic evidence of right ventricular dysfunction. The patient was successfully liberated from mechanical ventilation by the third hospital day and discharged on the fifth day with anticoagulation therapy.

A three-month follow-up showed complete normalization of right ventricular size and systolic function, with no signs of residual pulmonary hypertension.

Study Author Conclusions

The use of bedside echocardiography to guide the administration of a second bolus of alteplase during CPR for cardiac arrest due to massive pulmonary embolism can be beneficial, as demonstrated by the excellent outcome in this case.  

 

References:
[1] Mahboob HB, Denney BW. Double Bolus Alteplase Therapy during Cardiopulmonary Resuscitation for Cardiac Arrest due to Massive Pulmonary Embolism Guided by Focused Bedside Echocardiography. Case Rep Crit Care. 2018;2018:7986087. Published 2018 Mar 19. doi:10.1155/2018/7986087

 

Use of Thrombolytic Therapy During Cardiac Arrest: A Single-Center Experience

Design

Single-center, retrospective, cohort study

N= 26

Objective

To characterize the use of thrombolytic therapy during cardiac arrest and to evaluate the rate of return of spontaneous circulation (ROSC) in a real-world setting

Study Groups

All patients (n= 26)

Inclusion Criteria

Adult patients who received alteplase during cardiac arrest between 2010 and 2015 at a tertiary academic medical center

Exclusion Criteria

Not specified

Methods

Retrospective analysis of adult patients who received alteplase during cardiac arrest. The median dose of alteplase administered was 100 mg. Pulmonary embolism was confirmed prior to administration in 5 patients. 

Duration

2010 to 2015

Outcome Measures

Primary: Rate of return of spontaneous circulation (ROSC)

Secondary: Survival to hospital discharge, incidence of bleeding events

Baseline Characteristics  

All patients (n= 26)

Male 65%
Caucasian 89%
Median age, years 64
History of preexisting venous thromboembolism 5 patients
Receiving systemic anticoagulation 8 patients
Results  

All patients (n= 26)

ROSC achieved 65%
Survived to hospital discharge 2 patients
Bleeding events in survivors 2 patients
Adverse Events

Both surviving patients experienced a bleeding event.

Study Author Conclusions

Thrombolytic therapy is used infrequently for cardiac arrest management and should be considered on a case-by-case basis, particularly when there is a high suspicion of pulmonary embolism as the cause of arrest.

Critique

The study is limited by its small sample size and retrospective design, which may affect the generalizability of the findings. The lack of a control group and detailed exclusion criteria further limits the ability to draw definitive conclusions about the efficacy and safety of thrombolytic therapy during cardiac arrest.

 

References:
[1] Smith SE, Johnson DC. “Real-World” Application of Thrombolysis in Cardiac Arrest. Journal of Pharmacy Practice. 2018;33(3):267-270. doi:10.1177/0897190018799187

 

Successful alteplase bolus administration for a presumed massive pulmonary embolism during cardiopulmonary resuscitation

Design

Case report

Case presentation

A 54-year-old male patient who suffered a cardiopulmonary arrest presumed to be due to massive pulmonary embolism (PE). Following recent ankle surgery, the individual presented with syncope and rapidly deteriorated. Due to the patient's noncompliance with prescribed postoperative thromboprophylaxis, there was a high suspicion of PE.

After 40 minutes of advanced cardiac life support without results, a 50 mg bolus of alteplase was administered intravenously over two minutes based on empirical decision-making. This intervention led to the return of spontaneous circulation (ROSC) within six minutes. Subsequent imaging using computed tomographic angiography confirmed the presence of a pulmonary embolism, validating the empirical use of fibrinolytic therapy.

Study Author Conclusions

The choice of fibrinolytic therapy should be based on hospital availability, with prompt initiation of treatment and incorporation of an intravenous bolus. Resuscitation should be continued for at least 30 minutes, or until ROSC, after fibrinolytic initiation to allow time for the medication to work.

 

References:
[1] Prom R, Dull R, Delk B. Successful alteplase bolus administration for a presumed massive pulmonary embolism during cardiopulmonary resuscitation. Ann Pharmacother. 2013;47(12):1730-1735. doi:10.1177/1060028013508644

 

Pulmonary Embolism as Cause of Cardiac Arrest: Presentation and Outcome

Design

Retrospective study

N= 60

Objective

To analyze clinical presentation, diagnosis, therapy, and outcome of patients with cardiac arrest after pulmonary embolism (PE)

Study Groups

Thrombolysis (n= 21)

Non-thrombolysis (n= 21)

Inclusion Criteria

Patients with cardiac arrest caused by PE admitted to the emergency department from July 1, 1991, to June 30, 1999

Exclusion Criteria

Patients with contraindications to thrombolytic therapy, known underlying severe morbidity, and expected neurological damage after prolonged resuscitation efforts

Methods

Retrospective analysis of clinical presentation, diagnosis, therapy, and outcome. Thrombolytic treatment with 100 mg of recombinant tissue-type plasminogen activator (rt-PA) was administered in different schemes. Echocardiography, spiral CT, and ventilation-perfusion scan were used for diagnosis.

Duration

July 1, 1991, to June 30, 1999

Outcome Measures

Primary: Return of spontaneous circulation

Secondary: Survival to hospital discharge, survival at 6 months

Baseline Characteristics   Thrombolysis (n= 21)

Non-thrombolysis (n= 21)

Age, years 60 66
Initial ECG rhythm - PEA 63% 63%
Initial ECG rhythm - Asystole 32% 32%
Initial ECG rhythm - Ventricular fibrillation 5% 5%
Results   Thrombolysis (n= 21)

Non-thrombolysis (n= 21)

p-value
Return of spontaneous circulation 81% 43% 0.03
Survival to hospital discharge 10% 0% -
Survival at 6 months 10% 0% -
Adverse Events

Bleeding complications observed in 5 patients; rupture of liver in 2 cases; mediastinal bleeding in 1 patient.

Study Author Conclusions

Mortality related to cardiac arrest caused by PE is high. Echocardiography is supportive in determining PE as the cause of cardiac arrest. Thrombolysis should be attempted to achieve return of spontaneous circulation and potentially better outcomes.

Critique

The study highlights the challenges in diagnosing PE as a cause of cardiac arrest and the potential benefits of thrombolysis. However, the retrospective design and small sample size limit the generalizability of the findings. Additionally, the study lacks a control group for comparison of outcomes with other treatment modalities.

 

References:
[1] Kürkciyan I, Meron G, Sterz F, et al. Pulmonary Embolism as Cause of Cardiac Arrest: Presentation and Outcome. Arch Intern Med. 2000;160(10):1529–1535. doi:10.1001/archinte.160.10.1529