What evidence is available to support IV enoxaparin dosing? What is appropriate IV dosing of enoxaparin? Is there evidence for VTE or AFib populations using IV enoxaparin?

Comment by InpharmD Researcher

Intravenous enoxaparin administered as a 30-minute infusion is found to be a viable alternative to subcutaneous dosing in critically ill pediatric patients, with doses typically ranging from 1 to 1.5 mg/kg for VTE treatment. Studies show IV administration achieves therapeutic anti-Factor Xa levels with comparable efficacy and safety to the subcutaneous route. However, this evidence is primarily from pediatric VTE studies in intensive care settings. Additional data from adults seem to support this finding. However, data specifically supporting the use of IV enoxaparin for VTE or AFib in the general adult population appears to be limited.

Background

A 2024 systematic review and meta-analysis of 15 retrospective studies, randomized controlled trials (RCTs), and case series, evaluated individualized dosing strategies for enoxaparin in critically ill pediatric patients. Significant interindividual variability was identified in enoxaparin pharmacokinetics, with evidence suggesting higher initial doses may be necessary, particularly in neonates and infants, to achieve therapeutic anti-Xa levels. Intravenous (IV) administration was noted as an alternative to subcutaneous (SC) dosing, with numerous studies demonstrating comparable safety and efficacy when comparing IV infusions over 30 minutes and SC dosing in critically ill patients. However, another study did find that 56% of neonates experienced localized reactions at the site of the indwelling SC catheter when receiving enoxaparin for thrombosis treatment. [1]

The route of administration is often determined based on the severity of the condition, the patient’s stability, or the risk of drug interactions, with critically ill pediatric patients usually receiving SC dosing due to the ease and lower risk of infection. While the recommended prophylactic venous thromboembolism (VTE) treatment with enoxaparin in pediatrics ranges from 0.5 to 1.5 mg/kg/12 hours, the treatment of deep vein thrombosis (DVT) or pulmonary embolism is 1 to 1.5 mg/kg/12 hours. However, opposing evidence suggested that patients younger than one year of age require a higher initial treatment dose of 1.5 to 2.7 mg/kg/12 hours instead, although this was administered subcutaneously. The findings emphasize the importance of therapeutic drug monitoring and population pharmacokinetic modeling for optimizing enoxaparin dosing in this vulnerable population, underscoring the need for further prospective research to establish pediatric-specific guidelines. [1]

A 2014 retrospective study evaluated the pharmacodynamics, safety, and therapeutic utility of intravenous (IV) enoxaparin delivered as a 30-minute infusion in critically ill neonatal and pediatric patients. This study included 45 individuals, 15 cases receiving IV enoxaparin and 30 controls receiving subcutaneous (SC) enoxaparin, between January 2009 and June 2012. Key inclusion criteria required patients to have at least one anti-Factor Xa level measured, with therapeutic ranges defined as 0.3-0.5 U/mL for prophylaxis and 0.5-1 U/mL for treatment. Cases and controls were matched in a 1:2 ratio based on age and ICU location to standardize potential confounding variables. A 30-minute IV infusion of enoxaparin was utilized in response to concerns about impaired SC absorption due to edema or to mitigate procedural pain, particularly in smaller or critically ill children. IV enoxaparin demonstrated comparable pharmacodynamic efficacy to SC administration, as therapeutic anti-Factor Xa levels were achieved in 100% of the cases and controls. [2]

Although IV dosing required a higher mean initial dose (1.14 ± 0.38 mg/kg/dose) than SC dosing (0.85 ± 0.2 mg/kg/dose; p= 0.003), cases requiring dose adjustments achieved therapeutic levels significantly faster (mean of 2.7 ± 1.3 days) than controls (mean of 4.4 ± 1.4 days; p= 0.007). Notably, no adverse events related to bleeding, thrombosis, or hypersensitivity were reported in either group. Additionally, the study highlighted lower perceived pain and distress with IV administration and outlined potential benefits in patients with compromised SC absorption due to factors such as edema, peripheral vasoconstriction, or reduced regional blood flow. The findings suggest that 30-minute IV enoxaparin infusions may be a viable, effective, and potentially more comfortable alternative for anticoagulation in critically ill pediatric populations, warranting further investigation and prospective pharmacokinetic and pharmacodynamic analyses. [2]

References:

[1] Kanan M, Alotaibi NM, Anzan KB, et al. Systematic review and meta-analysis of individualized enoxaparin dose optimization in critically ill pediatrics: A path towards enhanced therapeutic outcomes. Pharmacy Practice 2024 Jan-Mar;22(2):2948.https://doi.org/10.18549/PharmPract.2024.2.2948
[2] Cies JJ, Santos L, Chopra A. IV enoxaparin in pediatric and cardiac ICU patients. Pediatr Crit Care Med. 2014;15(2):e95-e103. doi:10.1097/PCC.0000000000000049

Literature Review

A search of the published medical literature revealed 5 studies investigating the researchable question:

What evidence is available to support IV enoxaparin dosing? What is appropriate IV dosing of enoxaparin? Is there evidence for VTE or AFib populations using IV enoxaparin?

Level of evidence

C - Multiple studies with limitations or conflicting results  Read more→



Please see Tables 1-5 for your response.


 

IV Versus Subcutaneous Enoxaparin in Critically Ill Infants and Children: Comparison of Dosing, Anticoagulation Quality, Efficacy, and Safety Outcomes

Design

Retrospective, observational, single-center study

N= 110 patients (139 courses)

Objective

To compare dosing, anticoagulation quality, safety, and efficacy of intravenous (IV) versus subcutaneous (SC) enoxaparin in critically ill infants and children

Study Groups

Therapeutic

IV enoxaparin (n= 40)

SC enoxaparin (n= 56)

Prophylactic

IV enoxaparin (n= 23)

SC enoxaparin (n= 20)

Inclusion Criteria

Patients who received SC or IV enoxaparin for treatment or prevention of thromboembolism; had at least one anti-FXa level during the admission

Exclusion Criteria

Not disclosed

Methods

Data were taken from children admitted to the cardiac intensive care unit (ICU), pediatric intensive care unit (PICU), or neonatal ICU who received enoxaparin. Enoxaparin was dosed either twice daily SC or infused over 30 minutes IV per institutional protocol. Starting doses for infants < 2 months old were 1.8 mg/kg/dose for target anti-FXa of 0.5-1 U/mL (therapeutic dose) and 0.75 mg/kg/dose for target anti-FXa level of 0.1-3 U/mL (prophylactic dose). In children ≥2 months, starting doses were 1 mg/kg/dose and 0.5 mg/kg/dose for therapeutic and prophylactic doses, respectively. Anti-FXa levels were drawn 4-6 h (for IV, timed from infusion initiation) after ≥2 initial or adjusted doses. Any adjustment doses or selection of administration route were determined by treating physicians.

Duration

January 2014 to March 2016

Outcome Measures

Clinical efficacy, dosing and anticoagulation quality measurements, safety

Baseline Characteristics

 

IV enoxaparin (n= 40)

SC enoxaparin (n= 56)

 

Age, months (IQR)

Children ≤3 months

Children >3 months

 

2 (1 to 3)

8 (4 to 58)

 

1.0 (0.1 to 3.0)

23 (7 to 35)

 

Weight, kg (IQR)

Children ≤3 months

Children >3 months

 

3.5 (2.4 to 4)

7.4 (4.6 to 16.7)

 

3.4 (3.2 to 4.5)

12 (7 to 14.9)

 

Male

Children ≤3 months

Children >3 months

 

66%

65%

 

69%

66%

 

Duration enoxaparin given, days (IQR)

Children ≤3 months

Children >3 months

 

22 (5 to 56)

8 (5 to 28)

 

20 (14 to 37)

12 (7 to 27)

 

Abbreviations: IQR= interquartile range

Data specifically tabulated for therapeutic dose courses above.

Results

Endpoint

IV enoxaparin (n= 40)

SC enoxaparin (n= 56)

p-value

Time required to achieve target anticoagulation levels, days (IQR)

Children ≤3 months

Children >3 months

 

1 (1 to 4)

4 (2 to 23)

 

3 (1 to 5)

9 (4 to 19)

 

0.07

0.30

Proportion of patients who achieved target levels on initial dosing

Children ≤3 months

Children >3 months

 

55%

40%

 

40%

39%

 

0.35

1.00

Dose adjustments per day of therapy (IQR)

Children ≤3 months

Children >3 months

 

0.1 (0.1 to 0.3)

2 (0 to 3)

 

0.1 (0 to 0.2)

1 (0 to 2)

 

0.53

0.89

Major or clinically relevant nonmajor bleeding

Children ≤3 months

Children >3 months

 

5%

5%

 

10%

6%

 

1.00

1.00

Anticoagulation failure

Children ≤3 months

Children >3 months

 

0

0

 

7%

0

 

1.00

1.00

Data specifically tabulated for therapeutic dose courses above.

Adverse Events

See Results

Study Author Conclusions

IV enoxaparin infused over 30 minutes appears to offer a safe and effective option for parenteral anticoagulation in critically ill infants and children at risk for thromboembolism. The results of this study support the need for a larger, multicenter prospective study comparing IV with SC enoxaparin administration for the prevention and treatment of thromboembolism in the critically ill pediatric population.

InpharmD Researcher Critique

Doses, dosing adjustments, and the decision whether to administer IV or SC enoxaparin were determined by treating physicians, the latter of which was present in the IV cohort (patients were typically younger with lower baseline weight vs SC cohort). Use of a small sample size and retrospective data further limit findings.



References:

Diab YA, Ramakrishnan K, Ferrell B, et al. IV Versus Subcutaneous Enoxaparin in Critically Ill Infants and Children: Comparison of Dosing, Anticoagulation Quality, Efficacy, and Safety Outcomes*. Pediatric Critical Care Medicine. 2017;18(5):e207-e214. doi:10.1097/PCC.0000000000001126

 

Experience with Intravenous Enoxaparin in Critically Ill Infants and Children

Design

Retrospective, observational pediatric chart review

N= 7

Objective

To report the effects of intravenous (IV) enoxaparin in pediatric patients in the intensive care unit (ICU) to derive pharmacodynamic data to determine dosing and monitoring for IV or subcutaneous (SC) use

Study Groups

Enoxaparin IV (N= 7)

Inclusion Criteria

All pediatric patients receiving IV enoxaparin in the pediatric intensive care unit (ICU) at Children’s Medical Center Dallas between April 1, 2005 and March 31, 2006

Exclusion Criteria

Not disclosed

Methods

All included subjects' medical records were retrospectively reviewed to determine therapeutic doses when enoxaparin is administered through IV route for prophylaxis or treatment. Prior to IV treatment, five patients were initiated on SC enoxaparin but were switched to IV due to marked edema, while the remaining two had no prior SC administration. Patients aged <1 year received enoxaparin every 8 hours, while those >1 year received treatment every 12 hours. To evaluate and compare the anti-Xa levels drawn after different doses and dosing regimens, data was transformed by using the following equation: anti-Xa level (IU/mL)/(total daily dose of enoxaparin/kg).

Duration

April 1, 2005 to March 31, 2006

Outcome Measures

Change in anti-Xa levels; mean therapeutic dose for intravenous and subcutaneous dosing

Baseline Characteristics

Patients* Age Indication for Anticoagulation Duration of IV enoxaparin Maximum creatinine on IV enoxaparin, mg/dL Mean albumin on IV enoxaparin, g/dL
1 2 months Thrombosis-R subclavian, innominate and internal jugular veins 3 days 0.2 2.9
2 3 months Thrombosis-L transverse and sigmoid cerebral sinus, superior vena cava syndrome 26 days 0.9 3.3
3 18 days Thrombosis-L cardiac ventricle; then switched to prophylaxis 17 days 1.6 2.9
4 2 months Thrombosis-R cardiac atrium 34 days 0.8 3.1
5 28 months Prophylaxis 5 days 0.9 3.4
6 3 years Thrombosis-L innominate vein; ischemic stroke 67 days 2.6 3.0
7 4 months Prophylaxis 10 days 0.5 3.3

Abbreviation: L= left; R= right; IV= intravenous

*All patients had congenital heart defects

Results

 

Anti-Xa Level (U/mL)

Dose (mg/kg/day)

Anti-Xa/dose

Time level drawn, hr

0 to 1.9

2 to 3.9

4 to 5.9

6 to 8

 

0.68

0.46

0.77

0.13

 

4.67

3.67

5.93

5.98

 

0.19

0.15

0.16

0.03

  Therapeutic Dose Prophylactic dose*

Patient age

Overall, mg/kg/dose

<1 year, mg/kg/dose

>1 year, mg/kg/dose

 

-

2.4 ± 0.58

1.11 ± 0.13

 

0.93 ± 0.43

-

-

 

Enoxaparin IV for thrombus treatment (n= 5)

Thrombus treatment

Complete resolution

Partial resolution

Succumb to disease before re-evaluation

 

3

1

1

 

*Prophylactic dose was not stratified due to small sample size

- Peak: 1-2 hrs after administration (p= 0.1193)

- Subtherapeutic dose exhibited: At 6 to 8 hours for most patients

Adverse Events

Common Adverse Events: N/A

Serious Adverse Events: No bleeding complications occurred.

Percentage that Discontinued due to Adverse Events: N/A

Study Author Conclusions

Our data show that the pharmacodynamics of intravenous administration is different from subcutaneous administration and deserves further study.

InpharmD Researcher Critique

This study was limited by its small sample size and retrospective study design, limiting researchers from calculating pharmacodynamic properties directly. While this study did exhibit the safety and efficacy of IV enoxaparin, further studies are warranted for in-depth pharmacodynamic and pharmacokinetic information.



References:

Crary SE, Van Orden H, Journeycake JM. Experience with intravenous enoxaparin in critically ill infants and children. Pediatr Crit Care Med. 2008;9(6):647-649. doi:10.1097/PCC.0b013e31818d1920

Intravenous enoxaparin anticoagulation in percutaneous left atrial cardiac procedures
Design

Retrospective, non-controlled, single-centre study

N= 198

Objective To report the first experience using intravenous enoxaparin without anticoagulation monitoring in transcatheter structural heart interventions performed in the left atrium
Study Groups

LAA closure (n= 80)

PFO closure (n= 68)

ASD closure (n= 50)

Inclusion Criteria All consecutive and unselected patients who underwent percutaneous LAA, PFO or ASD closure at a tertiary care centre using IV enoxaparin anticoagulation from January 2006 to December 2016
Exclusion Criteria Patients on UFH or exposed to vitamin K antagonists with an international normalised ratio ≥2 at the time of the procedure
Methods

Patients received a single IV enoxaparin dose of 0.5 mg/kg. A second dose of 0.25 to 0.5 mg/kg was considered for prolonged interventions (≥1 hour). Catheter flushes used isotonic saline with enoxaparin at 6 IU/mL of anti-Xa. No coagulation monitoring was performed. Follow-up was scheduled at three months.

Duration January 2006 to December 2016
Outcome Measures Primary: Occurrence of in-hospital death, embolic complications (stroke, TIA, peripheral arterial embolism), and major bleedings (type 3a or more according to BARC definitions
Baseline Characteristics   Total LAA closure PFO closure ASD closure
Number of patients 198 80 68 50
Male gender, n (%) 109 (55%) 56 (70%) 33 (49%) 20 (40%)
Age (years) 60±18 74±9 51±17 50±16
Systemic hypertension 92 (46%) 68 (85%) 13 (19%) 11 (22%)
Diabetes 27 (14%) 24 (30%) 1 (1%) 2 (4%)
Stroke or TIA 100 (51%) 42 (53%) 48 (71%) 10 (20%)
Congestive heart failure 14 (7%) 8 (10%) 2 (3%) 4 (8%)
Results   Total (n=198)      
Primary composite endpoint 6 (3%)      
In-hospital death 1 (0.5%)      
Type 3a bleedings 4 (2%)      
Transient ischaemic attack 1 (0.5%)      
Adverse Events

The primary composite endpoint occurred in 3% of patients, including 2% type 3a bleedings, 0.5% transient ischaemic attack, and 0.5% death from sepsis. Use of more than one dose of enoxaparin tended to be associated with more severe bleeding complications.

Study Author Conclusions

IV enoxaparin without monitoring appears to be a potentially safe and easy-to-use anticoagulation regimen in percutaneous LA cardiac interventions. Further investigations with larger cohorts of patients are warranted.

Critique

The study is limited by its retrospective and non-controlled design, which may introduce bias. The sample size for each type of intervention is relatively small, limiting the generalizability of the findings. Despite these limitations, the study provides valuable exploratory data on the use of IV enoxaparin in structural heart interventions.

 

References:

Guedeney P, Hammoudi N, Duthoit G, et al. Intravenous enoxaparin anticoagulation in percutaneous left atrial cardiac procedures. EuroIntervention. 2017;13(10):1226-1233. doi:10.4244/EIJ-D-17-00518

Intravenous enoxaparin or unfractionated heparin in primary percutaneous coronary intervention for ST-elevation myocardial infarction: the international randomised open-label ATOLL trial
Design

Randomized open-label trial

N=910

Objective To compare traditional heparin treatment with intravenous enoxaparin in primary PCI for ST-elevation myocardial infarction
Study Groups

Enoxaparin (n=450)

Unfractionated heparin (n=460)

Inclusion Criteria Patients with STEMI older than 17 years with an indication for primary PCI within 12 hours of symptom onset, or between 12-24 hours with persistent symptoms or ST elevation, or with shock or cardiac arrest (<10 min)
Exclusion Criteria Patients who received any anticoagulant before randomization, administration of thrombolytic agents for the present episode, short life expectancy, childbearing potential, known contraindications to treatment with aspirin, thienopyridines, or heparins
Methods Patients were randomly assigned to receive an intravenous bolus of 0.5 mg/kg of enoxaparin or unfractionated heparin before primary PCI. The primary endpoint was 30-day incidence of death, complication of myocardial infarction, procedure failure, or major bleeding. The main secondary endpoint was the composite of death, recurrent acute coronary syndrome, or urgent revascularization. Analysis was by intention to treat.
Duration July 2008 to January 2010
Outcome Measures

Primary: 30-day incidence of death, complication of myocardial infarction, procedure failure, or major bleeding

Secondary: Composite of death, recurrent acute coronary syndrome, or urgent revascularization

Baseline Characteristics   Enoxaparin (n=450) Unfractionated heparin (n=460)
Age, median (years) 59 (52–71) 60 (52–70)
Women 97 (22%) 101 (22%)
Weight, kg 75.0 (67.0–85.0) 75.5 (67.0–86.5)
Present smoking 199 (44%) 218 (47%)
Diabetes mellitus, all 63 (14%) 69 (15%)
Hypertension 205 (46%) 207 (45%)
Previous MI 28 (6%) 44 (10%)
Previous PCI 33 (7%) 53 (12%)
Killip class II, III, or IV 35 (8%) 51 (11%)
Time from symptom onset to randomisation, median (min) 153 (89–290) 139 (86–277)
Aspirin 433 (96%) 439 (95%)
Clopidogrel, any 418 (93%) 428 (93%)
Glycoprotein IIb/IIIa inhibitors, any 347 (77%) 382 (83%)
Results   Enoxaparin (n=450) Unfractionated heparin (n=460) p-value
Death, complication of MI, procedure failure, or major bleeding (primary endpoint) 126 (28%) 155 (34%) 0.063
Death, recurrent MI or ACS, or urgent revascularisation (main secondary endpoint) 30 (7%) 52 (11%) 0.015
Death, complication of MI, or major bleeding (net clinical benefit) 46 (10%) 69 (15%) 0.030
Death or complication of MI 35 (8%) 57 (12%) 0.021
Death, recurrent MI, or urgent revascularization 23 (5%) 39 (8%) 0.044
Adverse Events Major bleeding was similar between groups (5% for both). Minor bleeding was slightly lower in the enoxaparin group (7% vs. 9%)
Study Author Conclusions Intravenous enoxaparin compared with unfractionated heparin significantly reduced clinical ischemic outcomes without differences in bleeding and procedural success, providing an improvement in net clinical benefit in patients undergoing primary PCI.
Critique

The study's open-label design and exclusion of patients who received anticoagulation before randomization may limit generalizability. However, it provides valuable insights into the efficacy of enoxaparin in a real-world setting with consistent anticoagulation strategies. The study was underpowered for low-frequency events, and the lack of a screening log limits the generalizability of the findings.

 

References:

Montalescot G, Zeymer U, Silvain J, et al. Intravenous enoxaparin or unfractionated heparin in primary percutaneous coronary intervention for ST-elevation myocardial infarction: the international randomised open-label ATOLL trial. Lancet. 2011;378(9792):693-703. doi:10.1016/S0140-6736(11)60876-3

Intravenous enoxaparin guided by anti-Xa in venovenous extracorporeal membrane oxygenation: A retrospective, single-center study
Design

Retrospective, single-center study

N= 38

Objective To demonstrate the feasibility of Enoxaparin anticoagulation for VV ECMO patients
Study Groups

All patients (n= 38)

Non-COVID19 (n= 15)

COVID19 (n= 23)

Inclusion Criteria VV ECMO support implanted due to respiratory failure in the ICU or Enoxaparin administered intravenously in a continuous form, strictly followed an anticoagulation protocol and were at least 18 years old
Exclusion Criteria Pregnancy, using double lumen cannula (Avalon), patients after thoraco-abdominal surgery, patients after lung transplantation in early postoperative periods, patients after trauma without any type of anticoagulation, “heparin free” ECMO and patients with fibrinolytic treatment
Methods Retrospective analysis of VV ECMO patients on continuous intravenous Enoxaparin anticoagulation. The anticoagulation protocol started with bolus administration of 0.5 mg/kg Enoxaparin intravenously before ECMO cannulation, followed by continuous intravenous Enoxaparin aiming for anti-Xa 0.5 ± 0.1 IU/mL. Anti-Xa activity was measured every 6 hours on the first day and then every 8 hours. The speed of the Enoxaparin infusion was adjusted accordingly
Duration May 2019 to August 2023
Outcome Measures

Primary: Incidence of bleeding, thrombotic, and neurological complications during ECMO support

Baseline Characteristics   All patients (n= 38) Non-COVID19 (n= 15) COVID19 (n= 23)
Age (years) 52 ± 12 47 ± 16 54 ± 9
Weight (kg) 93 ± 23 84 ± 34 99 ± 20
Height (cm) 174 ± 9 172 ± 10 176 ± 8
Males (n) 24 7 17
Females (n) 14 8 6
0 type blood group (n) 10 4 6
A type blood group (n) 20 9 11
B type blood group (n) 6 2 4
AB type blood group (n) 2 0 2
APACHE II score 14 ± 4 15 ± 5 13 ± 3
SOFA score 9 ± 2 9 ± 2 9 ± 2
Norepinephrine (ug/kg/min) 0.26 ± 0.25 0.39 ± 0.29 0.18 ± 0.2
Vasopressin (units/min) 0.01 ± 0.01 0.01 ± 0.02 0.001 ± 0.01
ECMO flow (L/min) 4.8 ± 0.9 4.5 ± 1.2 5 ± 0.6
Hb before ECMO (g/L) 119 ± 20 118 ± 17 119 ± 22
Hb after ECMO (g/L) 97 ± 9 93 ± 10 99 ± 8
pRBCs per one patient (units) 2.2 ± 2.1 2.2 ± 1.8 2.3 ± 2.4
PaO2/FIO2 ratio before ECMO 108 ± 28 108 ± 26 108 ± 30
PaO2/FIO2 ratio after ECMO 267 ± 62 251 ± 80 275 ± 51
Hypoxic type of respiratory failure (n) 10 3 7
Hypercapnic type of respiratory failure (n) 3 2 1
Global type of respiratory failure (n) 25 10 15
Bleeding complications (n) 2 0 2
Thrombotic complications (n) 1 0 1
Neurological complications (n) 4 1 3
Mortality during ECMO period (n) 4 1 3
Mortality during ICU hospitalization (n) 10 5 5
Results   All patients (n= 38) Non-COVID19 (n= 15) COVID19 (n= 23)
Bleeding complications 2 (5.3%) 0 2
Thrombotic complications 1 (2.6%) 0 1
Neurological complications 4 (10.5%) 1 3
Adverse Events Bleeding complications (5.3%), thrombotic complications (2.6%), neurological complications (10.5%)
Study Author Conclusions Enoxaparin anticoagulation appears to be feasible for VV ECMO patients without an increase in adverse events. Further larger-sampled and comparative studies are needed in the future to support our findings
Critique The study provides valuable insights into the feasibility of using Enoxaparin for VV ECMO patients, showing a low incidence of complications. However, the retrospective design and small sample size limit the generalizability of the findings. Additionally, the lack of a comparative arm with UFH limits the ability to directly compare the efficacy and safety of Enoxaparin against the standard treatment.
References:

Durila M, Vajter J, Garaj M, et al. Intravenous enoxaparin guided by anti-Xa in venovenous extracorporeal membrane oxygenation: A retrospective, single-center study. Artif Organs. 2025;49(3):486-496. doi:10.1111/aor.14879